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A bacterial metabolite induces glutathione-tractable proteostatic damage, proteasomal disturbances, and PINK1-dependent autophagy in C. elegans | Cell Death & Disease
Indeed, pathways highlighted by genetics (TSLP, IL-33/ST2, IgE regulation via IL-4/IL-13, etc.) correspond to many of the new targeted therapies being developed or already in use in asthma
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